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SELECTIVE MC3R/MC4R MELANOCORTIN AGONIST · RESEARCH HUB

PT-141 Research Hub — Bremelanotide Melanocortin Studies

PT-141 (bremelanotide) is a synthetic cyclic heptapeptide derived as a metabolite of Melanotan-II, with greater selectivity for the MC3R and MC4R melanocortin receptors. It is widely cited in central melanocortin pathway and behavioural-pharmacology research.

What this hub covers

  • Cyclic heptapeptide structure and metabolite origin from MT-2
  • MC3R/MC4R-selective melanocortin agonism
  • Central melanocortin pathway literature
  • Comparisons to non-selective MT-2 and α-MSH
  • Reconstitution, storage and handling for research use

PT-141 research articles

Wellness Research

Melanocortin Research Overview

The melanocortin system consists of five receptor subtypes (MC1R–MC5R), endogenous peptide ligands derived from proopiomelanocortin (POMC), and two endogenous antagonists (Agouti protein and Agouti-related protein, AgRP). POMC is a large...

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PT-141 research FAQ

What is PT-141?
PT-141 (bremelanotide) is a synthetic cyclic heptapeptide that activates the melanocortin receptors, with relatively greater selectivity for MC3R and MC4R than non-selective analogs like MT-2.
How does PT-141 differ from MT-2?
PT-141 is derived as a linear metabolite of MT-2 and shows reduced activity at MC1R (pigmentation) and increased relative MC3R/MC4R selectivity, making it a distinct research tool for central melanocortin pathway studies.
What receptors does PT-141 act on?
PT-141 is an agonist at MC1R, MC3R, MC4R and MC5R, with the strongest research focus on MC3R/MC4R-mediated central pathways. See the PT-141 research overview for the receptor selectivity breakdown.
How is PT-141 stored?
Lyophilized PT-141 is stored at 2–8 °C protected from light. Reconstituted material should be refrigerated and used within the COA's documented window.

All content on this hub is provided strictly for laboratory research purposes. Compounds listed are not for human or veterinary consumption. See our research-use disclosure for full terms.